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Murine models for pharmacological studies of the metabolic syndrome
03-P008 : Improvement of insulin sensitivity by imidazoline-like drugs in rats
CO-015 : Imidazoline I1 receptor ligands activate hepatic adiponectin pathways and thus improve insulin sensitivity
0260 : Imidazoline I1 receptor ligands activate hepatic adiponectin pathways and thus improve insulin sensitivity
0321 : Imidazoline-like drug improve insulin sensitivity through a direct stimulation of adiponectin secretion in primary adipocytes
CO-007 : Imidazoline-like drug improve insulin sensitivity through a direct stimulation of adiponectin secretion in primary adipocytes
11-02 : Effects of  imidazoline-like drugs on insulin sensitivity and adiponectin pathway
CASD 002 : Cardiac muscarinic receptor overexpression in sudden infant death syndrome
Sympathetic overactivity as an early manifestation of metabolic syndrome
Cardiac muscarinic receptor overexpression in sudden infant death syndrome
Cardiac muscarinic receptor overexpression as a possible cause of vagal hyperreactivity
0145 : Involvement of the sympathetic nervous system in the development of metabolic disorders
Synthesis and biological evaluation of 2-aryliminopyrrolidines as selective ligands for I1 imidazoline receptors: discovery of new sympatho-inhibitory hypotensive agents with potential beneficial effects in metabolic syndrome
129 : Cardiac muscarinic receptor overexpression as a possible cause of vagal hyperreactivity
Evidence for an improvement of glucose tolerance by activation of peripheral imidazoline receptors in rats
A new pyrroline compound selective for I1-imidazoline receptors improves metabolic syndrome in rats
308 : Cardiac muscarinic receptor overexpression in sudden infant death syndrome
Methylation of imidazoline related compounds leads to loss of alpha(2)-adrenoceptor affinity. Synthesis and biological evaluation of selective I(1) imidazoline receptor ligands
Imidazoline-like drugs improve insulin sensitivity through peripheral stimulation of adiponectin and AMPK pathways in a rat model of glucose intolerance

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