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Mechanisms for the Specific Properties of γ-Hydroxybutyrate in Brain.
A proposed preventive role for Gamma-hydroxybutyrate (XyremR) in Alzheimer’s disease
Alzheimer, mitochondria and gender
Beneficial effects of Gelsemium-based treatment against paclitaxel-induced painful symptoms
Social Isolation in Early versus Late Adolescent Mice Is Associated with Persistent Behavioral Deficits That Can Be Improved by Neurosteroid-Based Treatment
Sex hormone-related neurosteroids differentially rescue bioenergetic deficits induced by amyloid-β or hyperphosphorylated tau protein.
Xanthurenic acid is localized in neurons in the central nervous system.
Testosterone and estrogen in multiple sclerosis: from pathophysiology to therapeutics
Allopregnanolone and its analog BR 297 rescue neuronal cells from oxidative stress-induced death through bioenergetic improvement.
Behavioral and electromyographic assessment of oxaliplatin-induced motor dysfunctions: Evidence for a therapeutic effect of allopregnanolone.
Protective effect of 4-Phenylbutyrate against proteolipid protein mutation-induced endoplasmic reticulum stress and oligodendroglial cell death
Age-related vulnerability of pattern separation in C57BL/6J mice
An autophagy-targeting peptide to treat chronic inflammatory demyelinating polyneuropathies
5-HIAA induces neprilysin to ameliorate pathophysiology and symptoms in a mouse model for Alzheimer’s disease
Evidence for effective structure-based neuromodulatory effects of new analogues of neurosteroid allopregnanolone
Novel analogs of allopregnanolone show improved efficiency and specificity in neuroprotection and stimulation of proliferation.
Allopregnanolone and Progesterone in Experimental Neuropathic Pain: Former and New Insights with a Translational Perspective
FTY720 controls disease severity and attenuates sciatic nerve damage in chronic experimental autoimmune neuritis
Neurophysiological responses to unpleasant stimuli (acute electrical stimulations and emotional pictures) are increased in patients with schizophrenia.
The translocator protein ligand XBD173 improves clinical symptoms and neuropathological markers in the SJL/J mouse model of multiple sclerosis

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