Résultats de la recherche

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Abnormal base excision repair at trinucleotide repeats associated with diseases: a tissue-selective mechanism
What is gained or ‘lost in translation’ in Huntington’s disease
Tissue-dependent regulation of RNAP II dynamics: the missing link between transcription and trinucleotide repeat instability in diseases?
Régulations épigénétiques et plasticité cérébrale : vers de nouvelles thérapies dans les maladies neurodégénératives ?
Transcription elongation and tissue-specific somatic CAG instability
The nucleotide sequence, DNA damage location, and protein stoichiometry influence the base excision repair outcome at CAG/CTG repeats
Neuronal identity genes regulated by super-enhancers are preferentially down-regulated in the striatum of Huntington's disease mice

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