Résultats de la recherche

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Fonction endocrine du cœur
Mechanisms involved in increased plasma brain natriuretic peptide after heart transplantation.
Atorvastatin treatment reduces exercise capacities in rats: involvement of mitochondrial impairments and oxidative stress.
Pressure overload-induced mild cardiac hypertrophy reduces left ventricular transmural differences in mitochondrial respiratory chain activity and increases oxidative stress
The Emerging Role of Mitochondria in Inflammatory Myopathies
Mitochondrial uncoupling reduces exercise capacity despite several skeletal muscle metabolic adaptations
Angiotensin-converting enzyme inhibition prevents myocardial infarction-induced increase in renal cortical cGMP and cAMP phosphodiesterase activities
Skeletal Muscle Mitochondrial Function in Peripheral Arterial Disease: Usefulness of Muscle Biopsy
Impact of iron oxide nanoparticles on brain, heart, lung, liver and kidneys mitochondrial respiratory chain complexes activities and coupling.
Opposite effects of statins on mitochondria of cardiac and skeletal muscles: a 'mitohormesis' mechanism involving reactive oxygen species and PGC-1.
Methylene blue protects liver oxidative capacity after gut ischaemia-reperfusion in the rat.
Isoflurane anesthesia preserves liver and lung mitochondrial oxidative capacity after gut ischemia-reperfusion.
Left Ventricular Transmural Gradient in Mitochondrial Respiration Is Associated with Increased Sub-Endocardium Nitric Oxide and Reactive Oxygen Species Productions.
Pretreatment with brain natriuretic peptide reduces skeletal muscle mitochondrial dysfunction and oxidative stress after ischemia-reperfusion.
Effect of postconditioning on mitochondrial dysfunction in experimental aortic cross-clamping.
Mitochondria of trained skeletal muscle are protected from deleterious effects of statins.
Impairment of maximal aerobic power with moderate hypoxia in endurance athletes: do skeletal muscle mitochondria play a role?
Reductive stress impairs myoblasts mitochondrial function and triggers mitochondrial hormesis.
Skeletal muscle mitochondrial dysfunction during chronic obstructive pulmonary disease: central actor and therapeutic target.

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